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- W96687390 abstract "Atherosclerosis is the result of complex gene/environment interactions and include disorders of the lipoprotein system [1]. Well intentioned focus on the detection and treatment of elevated LDLC has resulted in some reduction in cardiovascular events, but despite successful reductions in LDLC, atherosclerosis continues to progress in many patients [2]. An approximate 25% reduction in clinical cardiovascular events has been reported from many trials of LDLC reduction (Figure 1). This is a statistically significant reduction in clinical events but it can be misleading since it masks the fact that many patients receiving cholesterol lowering medications, continue to have CV events. Clearly, attention only to LDLC reduction ignores the multiple other metabolic contributors to atherosclerosis, one of which is oxidative damage of lipoprotein particles and the resultant cascade of issues that promote atherosclerosis either independent of LDL or in combination with LDL [3,4]." @default.
- W96687390 created "2016-06-24" @default.
- W96687390 creator A5061619086 @default.
- W96687390 date "2000-01-01" @default.
- W96687390 modified "2023-09-27" @default.
- W96687390 title "Lipoproteins and Oxidation: Clinical Aspects of Lipoprotein Metabolism and Oxidation" @default.
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- W96687390 doi "https://doi.org/10.1007/978-94-011-4375-2_1" @default.
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