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- W97024277 abstract "297 Objectives To assess myocardial blood flow (MBF) and coronary vasodilator reserve using N-13 ammonia PET in subjects with cardiac amyloidosis and to compare them to normal subjects and to subjects with left ventricular hypertrophy. Amyloid deposits in the heart could compress the microvasculature, resulting in coronary microvascular dysfunction and angina, even in the absence of epicardial coronary artery disease (CAD). Methods We prospectively enrolled 24 subjects in this study. A group of healthy subjects (Normal, N=10) and subjects with hypertensive left ventricular hypertrophy (LVH, N=5) were recruited as controls. Ten subjects had biopsy-proven light chain (N=7) or senile (N=3) amyloidosis (Amyloid), with typical cardiac involvement on echocardiography. Exclusion criteria included, EF Results The results are summarized in the Table and Figure. The MBF at rest was similar between the groups, but, peak stress MBF was significantly lower in the amyloid (0.91) compared to normal (1.94) and LVH subjects (2.1), P Conclusions Subjects with cardiac amyloidosis demonstrate significantly lower peak stress MBF and vasodilator coronary flow reserve compared to normal volunteers and subjects with LVH. Research Support Amyloidosis Foundation; American Society of Nuclear Cardiology, NIH-NHLB" @default.
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- W97024277 date "2012-05-01" @default.
- W97024277 modified "2023-09-28" @default.
- W97024277 title "Coronary microvascular function in cardiac amyloidosis" @default.
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