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- W973333897 endingPage "2689" @default.
- W973333897 startingPage "2684" @default.
- W973333897 abstract "急性胰腺炎（acute pancreatitis，AP）是一种常见的急重症，经研究证实多种细胞因子的活化与AP的发生进展关系密切．而Toll样受体4（Toll-like receptor 4，TLR4）、核因子-κB（nuclear factor kappa B．NF-κB）与这些细胞因子的活化密切相关．他们在AP发生发展中作为介导炎症反应的枢纽起着重要作用．以往认为TLR4触发细胞内信号传导通路，从而激活NF-κB，调控大量细胞因子释放，介导炎性反应．目前对TLR4／NF-κB信号通路具体的传导路径及参与因子又有了新的认识，本文概述了TLR4／NF-κB信号通路，且概述了TLR4／NF-κB信号通路相关下调因子、增强因子，及能激活NF-κB的其他因子．" @default.
- W973333897 created "2016-06-24" @default.
- W973333897 creator A5003551778 @default.
- W973333897 creator A5051629727 @default.
- W973333897 date "2007-09-08" @default.
- W973333897 modified "2023-09-24" @default.
- W973333897 title "TLR4, NF-κB与急性胰腺炎" @default.
- W973333897 doi "https://doi.org/10.11569/wcjd.v15.i25.2684" @default.
- W973333897 hasPublicationYear "2007" @default.
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