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• W981627199 abstract "There is growing evidence that the development of some forms of mammalian associative conditioning depends on interactions between acetylcholine (ACh) and glutamate (GLUT) in neocortical neurons. Unlike long-term potentiation (LTP), another model of mammalian neural plasticity, this mechanism of enhancement appears to be based on postsynaptic changes in neural excitability induced by ACh, and a close relationship has been demonstrated between these changes and behavioral conditioning. Recent studies have shown that an end point of the ACh-mediated cascade is a decrease in a rapidly activated neuronal current. Single electrode voltage clamp techniques were adapted to measure currents in vivo in single cortical neurons. This chapter describes some of the evidence for involvement of ACh and GLUT in the development of conditioning, reviews evidence for a role of a rapidly activated potassium conductance, gK+(A), in mediating increased postsynaptic neural excitability without the need for steadily increased rates of baseline discharge and addresses a new issue, the possible role of GLUT in influencing the rate of conditioning." @default.
• W981627199 created "2016-06-24" @default.
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• W981627199 date "1993-01-01" @default.
• W981627199 modified "2023-10-14" @default.
• W981627199 title "Chapter 44: Cholinergic and glutamatergic effects on neocortical neurons may support rate as well as development of conditioning" @default.
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• W981627199 doi "https://doi.org/10.1016/s0079-6123(08)62420-6" @default.
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