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- W98223208 abstract "The acute respiratory distress syndrome (ARDS) is a common, highly morbid (and often fatal) critical illness characterized by the sudden onset of noncardiogenic pulmonary edema and respiratory failure. ARDS can occur in response to a direct pulmonary insult (e.g., pneumonia) or an indirect, systemic inflammatory condition (e.g., nonpulmonary sepsis). Forty years of intense investigation into the mechanisms underlying ARDS have fueled a revolution in the practice of critical care medicine, yielding improvements in supportive care such as lung-protective modes of ventilation. Despite these advances, there remains no clinically efficacious therapy targeted against ARDS pathophysiology. The failure to identify an efficacious mechanistic intervention in lung injury may arise from the incredible complexity of interrelated pathophysiological processes dynamically occurring within the injured alveolus. Increasing attention has thus been paid to determining the processes of ARDS recovery, in the hope that endogenous mechanisms of injury resolution could be therapeutically exploited." @default.
- W98223208 created "2016-06-24" @default.
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- W98223208 date "2014-01-01" @default.
- W98223208 modified "2023-09-27" @default.
- W98223208 title "Pathobiology of the Acute Respiratory Distress Syndrome" @default.
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- W98223208 doi "https://doi.org/10.1016/b978-0-12-386456-7.05309-0" @default.
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