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- W98588045 abstract "This chapter discusses acute stress and trace element metabolism. While considering the effect of acute stress upon the metabolism of a trace element, one must consider the fundamental physiological role of the element, methods available for its analysis, and the status of general and specific trace-metal nutrition in the host. Within cells, magnesium is related functionally to the activity of enzyme systems, especially those that generate high-energy phosphate bonds and control mitochondrial oxidative phosphorylation. Magnesium is a necessary cofactor of DNA, RNA, and ribosomes. It is needed for the binding of mRNA to polysomal subunits and for the activation of aminoacyl-sRNA complexes during protein synthesis. The extracellular concentration of magnesium ions influences both the nerve-cell conduction and myoneural junction activity. Excessive magnesium ions can produce local or general anesthesia, asystole, and curare-like paralysis. Deficiency of magnesium produces a syndrome of central nervous system irritability. The primary functional role of iron is oxygen transport via hemoglobin. Iron further serves as an essential cofactor for a number of heme and non-heme metalloenzymes that are important for oxygen and hydrogen exchange or electron transport. Iron also plays a role in maintaining the structure and integrity of polyribosomes and is important in protein synthesis. Zinc is an integral constituent and cofactor of more than twenty metalloenzymes. Because of relatively high concentrations of zinc found in various endocrine glands, this element may influence the secretion of several hormones. The important role of zinc in promoting the healing of thermal and surgical wounds adds further support to this hypothesis." @default.
- W98588045 created "2016-06-24" @default.
- W98588045 creator A5032082255 @default.
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- W98588045 date "1972-01-01" @default.
- W98588045 modified "2023-10-16" @default.
- W98588045 title "ACUTE STRESS AND TRACE ELEMENT METABOLISM" @default.
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