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- W990933150 endingPage "258" @default.
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- W990933150 abstract "The notion that the environment in which a fetus develops is critical for long-term health and optimal developmental outcome is well supported by research. The adaptation of the pregnant mother to psychological challenges or threats to homeostasis can have adverse effects on the offspring's development. Moreover, the pathways that link prenatal stress to suboptimal development in the offspring remain uncertain. Recent data suggest that stressful experiences in the life of the pregnant mother may be incorporated into the offspring's biology by altering both neural and hormonal processes. This chapter reviews the evidence establishing a relationship between prenatal stress, birth outcomes, and child behavior problems in humans. It describes the evidence that prenatal stress compromises birth weight, infant neurobehavior, behavioral/hypothalamic-pituitary-adrenal (HPA) axis reactivity to challenge, response to sensory stimulation, and dopaminergic function in rhesus monkey offspring. The effects found in the primate studies include reduced birth weight, altered HPA axis regulation, enhanced sensitization to tactile stimuli, and altered dopaminergic system function. Early gestation appears to be a sensitive period for these effects, and some of the effects seem to be compounded by other factors including exposure to alcohol consumption during pregnancy." @default.
- W990933150 created "2016-06-24" @default.
- W990933150 creator A5037061171 @default.
- W990933150 creator A5040242794 @default.
- W990933150 creator A5046243960 @default.
- W990933150 creator A5048727419 @default.
- W990933150 date "2008-01-01" @default.
- W990933150 modified "2023-10-16" @default.
- W990933150 title "Prenatal Stress Influences on Neurobehavior, Stress Reactivity, and Dopaminergic Function in Rhesus Macaques" @default.
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