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• W995102789 abstract "This chapter describes the biology of HIV and its abundant genetic variations. Point mutation classifies viruses into two classes depending on whether replication errors are corrected (DNA viruses) or not (retroviruses and RNA viruses). Mutation rates are expressed as the probability of making a point mutation per base and per round of replication. The fixation rate refers to the number of amino acid changed, accumulated, or fixed per year. There are considerable qualitative and quantitative differences between HIV and influenza A with respect to their mutation and recombination rates. HIV replicates in CD4+ T lymphocytes, dendritic cells, and macrophages, but cross-sectional quantitative analyses show that T lymphocytes carry the greatest viral burden. HIV is more abundant in memory cells as opposed to naive T cell compartments. The chapter focuses principally on these memory cells. Two complementary approaches are applied to the study of viral dynamics in vivo. The first and most widely used approach is an experimental classic system in which the HIV plasma viraemia is remarkably stable over time, varying little from one day to another, and the decay of viraemia can be measured and modeled using multiple anti-HIV drugs. The second approach involves the rates of fixation of mutations of viruses." @default.
• W995102789 created "2016-06-24" @default.
• W995102789 creator A5013201108 @default.
• W995102789 date "2003-01-01" @default.
• W995102789 modified "2023-10-18" @default.
• W995102789 title "HIV Variation—A Question of Signal-to-Noise" @default.
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• W995102789 doi "https://doi.org/10.1016/b978-012194851-1/50027-5" @default.
• W995102789 hasPublicationYear "2003" @default.
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