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- W996613263 abstract "In Brief The overwhelming body of research on T regulatory cells (Treg) has focused on CD4 + CD25 + Foxp3+ T cells. However, recent years have witnessed a resurgence in interest in CD4 − CD8+, CD4 − CD8− (double negative [DN]), and CD4 + Foxp3− type 1 Treg (Tr1) Treg and their role in controlling autoimmune diseases and in promoting the survival of organ allografts and xenografts. CD8+ and DN Treg can arise spontaneously (natural Treg) or can be induced in situ. Both CD8+ and DN Treg have been shown to enhance the survival of organ allografts and xenografts. Additionally, both can suppress alloimmune responses by contact-dependent mechanisms by either inducing apoptosis or mediating direct cytolysis of effector T cells. CD8+, DN, and Tr1 Treg can also act in a contact-independent manner by elaborating soluble immunosuppressive factors, such as TGF-β and IL-10. Applying CD8+, DN, and Tr1 Treg for enhancing the survival of organ allografts and xenografts is still in its infancy but holds significant potential. Furthermore, there is a need for a more comprehensive understanding of how current immunosuppressive therapies applied to organ transplantations affect the wide array of Treg populations. Ligocki and Niederkorn provide a timely review on the role of non-canonical FoxP3-negative regulatory T cells. Defining the role of these cells in transplantation is at its infancy but tantalizing data suggest that these cells may have significant therapeutic potential." @default.
- W996613263 created "2016-06-24" @default.
- W996613263 creator A5019891297 @default.
- W996613263 creator A5082183641 @default.
- W996613263 date "2015-08-01" @default.
- W996613263 modified "2023-10-14" @default.
- W996613263 title "Advances on Non-CD4 + Foxp3+ T Regulatory Cells" @default.
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